Low live weight heifers at 15 months are low live weight
compared to the rest of the herd until at least their third lactation.
The next question this draws up is where should the mature
live weight be drawn from? Should it be the genetic potential mature weight or
the average herd live weight? The herd's mature weight could, therefore, be a
function of historical heifer growth trajectory and projecting heifer growth on
data from cows that were poorly grown as heifers would perpetuate the cycle of
poor productivity.
40% calves suffer from failed passive transfer of
immunoglobulins caused by a failure to feed enough colostrum quickly enough to
the new born calf. Calves with FTP will have increased rates of mortality and
morbidity. It also reduces DM intake and milk production later on.
It is often reported anecdotally that graziers receiving
calves at weaning that the 'tail end calves' never do very well and will always
be behind the rest of the mob. This paper recognises accelerated ADG in the
first eight weeks is associated with enhanced mammary development and
identifies further work is required to understand the relationship between
early life average daily gain and gastro-intestinal development.
It is interesting
the way that different speeds of average daily gain has a different effect on
mammary development at different stages of the heifers path to maturity. The
links showing in the papers reviewed that an accelerated ADG pre wean has a
positive impact on mammary development and subsequent lactation after the first
calving. Then some studies will show a negative correlation between accelerated
growth and mammary development from post weaning through to puberty.
It is hard to
achieve a linear ADG in the grazed pasture growth system. This is due to the
high NDF levels in summer restricting the VFI and reducing the ME of the
pasture available to the heifer and low growth rates in the winter reducing the
level of feed available. The question is can the pastoral system be manipulated
to match the needs of the heifer and use the natural pastoral growth cycle to
feed the heifer to her own benefit and use compensatory growth to accelerate
the ADG when required?
Though stair step
methods of gain using compensatory growth have been shown to have poor utilisation
of energy for live weight gain and was used for lay down of fatty tissue rather
than lean tissue.
J. R. Roche, N. A. Dennis, K. A. Macdonald, C. V. C. Phyn,
P. R. Amer, R. R. White and J. K. Drackley (2015) Growth targets and rearing
strategies for replacement heifers in pasture-based systems: a review, Animal Production Science, 55(7),
902-915
Conclusions and further research
Mature Lwt is hard to define, because it is dependent on
both the genotype of the heifer and the environment in which she is managed.
Furthermore, there does not appear to be a significant advantage to defining a
mature Lwt for individual heifers compared with an average mature Lwt for the
herd. Although accepted as dogma that heifers should calve between 80% and 90%
of mature Lwt, the advantage of achieving this target is not clear. The effects
of pre-pubertal and post-pubertal ADG on milk production and reproduction are
inconsistent, as long as the heifer has had sufficient ADG to reach puberty
before the desired breeding date. That said, there is morphological and
physiological evidence that accelerated pre-pubertal ADG reduces mammary
parenchymal development; what is not clear, however, is whether, or not, there
is compensatory development of the mammary gland during the gestational
allometric development phase. What is also not clear is whether accelerated
growth trajectories result in changes to the composition of nonmammary tissue
and whether this could potentially have negative effects in the calved heifer.
Further research is required in these areas. Consideration should also be given
to the trajectory of gain, whether periods of restriction and re-alimentation
increase the amount and activity of mammary secretory tissue and whether the
advantage can be exploited in pasture-based systems. Further work is required
to understand why such a high proportion of calves fail to receive sufficient
high-quality colostrum to ensure adequacy of immunoglobulin transfer. The
importance of colostrum in the lifetime productivity of the replacement animal
has been known for decades. Therefore, the failure of this vital practice on
farm is worthy of social study and increased extension effort. A growing area
of interest is the pre-weaning nutrition of the calf and, in particular, the
first 8 weeks of life. Research has indicated a significant return on limited
additional investment during this time. However, the majority of that work has
been undertaken in high-production, housed dairy systems. Further research is
required in grazing systems and, in particular, research efforts should focus
on determining whether the window of opportunity is even shorter than has been
indicated.
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